Need Help?

The Jerusalem Perinatal Study (JPS) aimed to examine the developmental origins of cardiometabolic risk.

The Jerusalem Perinatal Study (JPS) aimed to examine the developmental origins of cardiometabolic risk and explore the mechanisms underlying the associations between perinatal characteristics and the development of risk for adult chronic disease. In other words, the cohort has enabled the examination of associations between maternal and paternal characteristics with cardiometabolic risk variables in offspring at various points in the life course, as well as the effect of longitudinal changes in characteristics in both parents and offspring.

Click on a Dataset ID in the table below to learn more, and to find out who to contact about access to these data

Dataset ID Description Technology Samples
EGAD00010001816 Affymetrix Biobank array 2714
Publications Citations
Cohort Profile: The Jerusalem Perinatal Family Follow-Up Study.
Int J Epidemiol 45: 2016 343-352
9
Parent-of-Origin Effects of the APOB Gene on Adiposity in Young Adults.
PLoS Genet 11: 2015 e1005573
11
Associations of socioeconomic position in childhood and young adulthood with cardiometabolic risk factors: the Jerusalem Perinatal Family Follow-Up Study.
J Epidemiol Community Health 71: 2017 43-51
10
Accounting for Life-Course Exposures in Epigenetic Biomarker Association Studies: Early Life Socioeconomic Position, Candidate Gene DNA Methylation, and Adult Cardiometabolic Risk.
Am J Epidemiol 184: 2016 520-531
18
Maternal gestational weight gain and DNA methylation in young women: application of life course mediation methods.
Epigenomics 9: 2017 1559-1571
1
Associations of social environment, socioeconomic position and social mobility with immune response in young adults: the Jerusalem Perinatal Family Follow-Up Study.
BMJ Open 7: 2017 e016949
4
Mode of delivery and offspring adiposity in late adolescence: The modifying role of maternal pre-pregnancy body size.
PLoS One 14: 2019 e0209581
3
Searching for parent-of-origin effects on cardiometabolic traits in imprinted genomic regions.
Eur J Hum Genet 28: 2020 646-655
5